
Saxitoxin - Wikipedia
Saxitoxin (STX) is a potent neurotoxin and the best-known paralytic shellfish toxin. Ingestion of saxitoxin by humans, usually by consumption of shellfish contaminated by toxic algal blooms, is responsible …
Saxitoxin: A Comprehensive Review of Its History, Structure ...
Saxitoxin (STX) is a potent toxin produced by marine dinoflagellates and freshwater or brackish water cyanobacteria, and is a member of the paralytic shellfish toxins (PSTs).
Saxitoxin - an overview | ScienceDirect Topics
Saxitoxin Saxitoxin (STX) is a neurotoxin produced by some marine dinoflagellates that can be accumulated in shellfish during toxic algal blooms known as Red Tide.
Scalable total synthesis of saxitoxin and related natural ...
Aug 26, 2025 · Saxitoxin (STX, 1), a potent neurotoxin from shellfish, first isolated in 1957 (ref. 1), offers immense pharmaceutical potential owing to its interaction with voltage-gated sodium channels 2,...
Individuals handling saxitoxin or working in a laboratory where saxitoxin is being handled must receive training about potential risks from exposure to the toxin as well as safe handling of the toxin as …
Saxitoxin: A Toxin That Causes Paralytic Shellfish Poisoning
Jul 22, 2025 · Saxitoxin is a neurotoxin from marine algae that causes Paralytic Shellfish Poisoning by disrupting the communication between nerve cells.
Saxitoxin - American Chemical Society
Oct 23, 2023 · Saxitoxin takes its name from the Alaskan butter clam (Saxidomus gigantea) from which it was first identified in 1937 by Hermann Sommer 1 and co-workers at the University of California, …
An Overview on the Marine Neurotoxin, Saxitoxin: Genetics ...
Saxitoxin and its derivatives, collectively referred to as paralytic shellfish toxins (PSTs), are unique among neurotoxins in that they are found in both marine and freshwater environments by organisms …
Saxitoxin | C10H17N7O4 | CID 56947150 - PubChem
Saxitoxin is a carbamate alkaloid neurotoxin isolated from marine dinoflagellates and cyanobacteria that inhibits nerve conduction by blocking neuronal voltage-gated sodium channels in excitable cells.